Alexander Kikuchi, MD, PhD

Assistant Professor (Clinical), University of Utah School of Medicine

Specialties

• Gastrointestinal and hepatobiliary pathology
• Breast pathology

Education

• Medical Degree—University of Pittsburgh School of Medicine
• Doctorate Degree—University of Pittsburgh School of Medicine
• Fellowship—Gastrointestinal and Hepatobiliary Pathology, University of California San Francisco
• Fellowship—Surgical Pathology, University of California San Francisco
• Residency—Pathology, University of California San Francisco
• Bachelor of Arts, Bachelor of Music—University of Southern California

Certification/Affiliations

• American Board of Pathology (Anatomic Pathology)
• Hans Popper Hepatopathology Society
• Rodger C. Haggit Gastrointestinal Pathology Society
• Pancreatobiliary Pathology Society

Research Interests

• Histopathologic and molecular classification of primary liver neoplasms
• Chronic liver injury, hepatic fibrosis, and hepatic carcinogenesis

Awards

• Hans Popper Hepatopathology Society Best Trainee Abstract Award (1st place) at 2022 USCAP Meeting
• Robert E. Lee, MD, Award for Excellence in Anatomic Pathology (2019, University of Pittsburgh School of Medicine)
• Presidential Poster Award at 2017 AASLD Conference
• Summa Cum Laude (2010, University of Southern California)
• Renaissance Scholar (2010, University of Southern California)

Recent Publications

• Kikuchi AT, Umetsu S, Joseph N, et al. Genomic analysis in the categorization of poorly differentiated primary liver carcinomas. Am J Surg Pathol. 2023;47(11):1207-1218.
• Kikuchi A, Singh K, Gars E, et al. Pathology updates and diagnostic approaches to haemophagocytic lymphohistiocytosis. Histopathology. 2022;80(4):616-626.
• Kikuchi A, Singh S, Poddar M, et al. Hepatic stellate cell-specific platelet-derived growth factor receptor-α loss reduces fibrosis and promotes repair following hepatocellular injury. Am J Pathol. 2020;190(10):2080-2094.
• Zhang R, Kikuchi AT, Nakao T, et al. Elimination of Wnt secretion from stellate cells is dispensable for zonation and development of liver fibrosis following hepatobiliary injury. Gene Expr. 2019;19(2):121-136.